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Saturday, May 2, 2020 | History

3 edition of Sequestration of L-cell potential for DNA and protein synthesis by the meningopneumonitis agent found in the catalog.

Sequestration of L-cell potential for DNA and protein synthesis by the meningopneumonitis agent

Jennifer Jean Alexander

Sequestration of L-cell potential for DNA and protein synthesis by the meningopneumonitis agent

  • 217 Want to read
  • 25 Currently reading

Published .
Written in English


Classifications
LC ClassificationsMicrofilm 20808
The Physical Object
FormatMicroform
Paginationiv, 61 p.
Number of Pages61
ID Numbers
Open LibraryOL1249585M
LC Control Number94895132


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Sequestration of L-cell potential for DNA and protein synthesis by the meningopneumonitis agent by Jennifer Jean Alexander Download PDF EPUB FB2

The realization that the cellular transcription factor E2F is a target for the action of the retinoblastoma tumor suppressor protein, as well as related regulatory proteins, and that this regulation is disrupted by oncogenic products of the DNA tumor viruses, has led to studies that have provided important insights into molecular mechanisms of cellular growth by: There is an urgent need to develop effective vaccines against pneumonic plague, a highly lethal and contagious disease caused by the Gram-negative bacterium Yersinia we demonstrate that a novel DNA vaccine expressing a modified V antigen (LcrV) of Y.

pestis, with a human tissue plasminogen activator (tPA) signal sequence, elicited strong V-specific antibody responses in BALB/c by: The cyclic GMP-AMP synthase (cGAS) protein is a pattern-recognition receptor of the mammalian innate immune system that is recognized as a main cytosolic sensor of pathogenic or damaged DNA.

cGAS. Neeper MP, Kuo LM, Kiefer MC, Robb RJ. Structure function relationships for the IL 2 receptor system. III. Tac protein missing amino acids to (exon 4) is unable to bind IL 2: detection of spliced protein after L cell transfection.

J Immunol. May 15; (10)–Cited by: 5. The effect of 8-OH-DPAT was prevented by the 5-HT1A receptor antagonists, NAN and WAY, at doses without any intrinsic effect.N-ethoxycarbonylethoxy-1,2-dihydroquinoline (EEDQ 10 mg kg−1), an alkylating agent that inactivates different G-protein coupled receptors, impaired retention performance when given 48 h pretraining.

The.